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. 2017 Sep 7;7:10902. doi: 10.1038/s41598-017-10787-z

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Specific inhibition of host physiology after SpltNPV infection. (a) KEGG pathway enrichment across constant and down-regulated Sf21 proteins at late stages of viral infection. Ribosomal proteins were over-represented in down-regulated category by 18-folds in comparison to the unchanged group while Base-excision repair proteins were 5-times more prevalent in constant category. Depiction of down-regulated components of (b) Ribosomal machinery, (c) Oxidative phosphorylation which represent highly afflicted KEGG pathways at late stages of SpltNPV infection. The proteins which were found to be down-regulated in TMT labelling analysis are shown in green. Pathway maps were generated using KAAS^58–61.