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. 2017 Aug 28;2017:6807452. doi: 10.1155/2017/6807452

Table 2.

Association between PTENP1 gene polymorphisms and risk of gastric cancer (rs7853346).

Genotype Cases N (%) Controls N (%) Crude OR (95% CI) P Adjusted OR (95% CI) P P c
Overall 768 768
Additive model 0.79 (0.67–0.94) 0.007 0.80 (0.67–0.95) 0.011 0.033
Codominant model
 CC 496 (64.6) 447 (58.2) 1 1
 CG 241 (31.4) 277 (36.1) 0.78 (0.63–0.97) 0.026 0.79 (0.63–0.98) 0.033 0.099
 GG 31 (4.0) 44 (5.7) 0.63 (0.39–1.02) 0.060 0.65 (0.40–1.06) 0.087 0.261
Dominant model
 CC 496 (64.6) 447 (58.2) 1 1
 CG+GG 272 (35.4) 321 (41.8) 0.76 (0.62–0.94) 0.010 0.77 (0.62–0.95) 0.015 0.045
P trend 0.013 0.039
Recessive model
 CC+CG 737 (96.0) 724 (94.3) 1
 GG 31 (4.0) 44 (5.7) 0.69 (0.43–1.11) 0.124 0.71 (0.44–1.14) 0.153 0.459
Allele
 C 1233 (79.8) 1171 (76.2) 1
 G 303 (20.2) 365 (23.8) 0.79 (0.66–0.94) 0.007
HWE 0.900

Abbreviations: OR, odds ratio; CI, confidence interval; HWE, Hardy-Weinberg expectations. Adjusted for age, sex, smoking status, drinking status, residence, hypertension, diabetes, and family history of GC in the logistic regression model. Pc after Bonferroni's correction. P trend for CC, CG, GG, and CG+GG genotypes. The significant results are in bold.