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. 2017 Sep 11;8:499. doi: 10.1038/s41467-017-00547-y

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 10 — Model for Cas5-mediated coupling of cell cycle progression to cell wall stress response. Cas5 is dephosphorylated by the protein phosphatase Glc7 in response to cell wall stress. Once dephosphorylated, it can translocate to the nucleus to govern the expression of a myriad of genes, including those important for cell cycle regulation and cell wall stress response. Cas5 regulates changes in gene expression through both Swi4/Swi6-dependent and independent mechanisms