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. 2017 Jul 24;14(4):4011–4020. doi: 10.3892/ol.2017.6659

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Copyright: © Yun et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — Representative images of immunohistochemical staining for COUP-TFII, LXR, RXRα, and SREBP-1c in colorectal cancer tissue. (A) Left, COUP-TFII-positive colorectal cancer tissue. Right, COUP-TFII-negative colorectal cancer tissue. (B) Left, LXR-positive colorectal cancer tissue. Right, LXR-negative colorectal cancer tissue. (C) Left, strong RXRα immunoreactivity detected in well-differentiated colorectal cancer tissues. Right, weak RXRα immunoreactivity detected in poorly differentiated colorectal cancer tissues. (D) Left, SREBP-1c-positive colorectal cancer tissue. Right, SREBP-1c-negative colorectal cancer tissue. Magnification, ×200. COUP-TFII, chicken ovalbumin upstream promoter-transcription factor II; LXR, liver X receptor; RXRα, retinoid X receptor α; SREBP-1c, sterol regulatory element binding protein-1c.