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. 2017 Sep 11;18:718. doi: 10.1186/s12864-017-4058-y

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — Non-canonical target sites have significantly more overlaps with heterochromatin sites. For both embryonic and maternal transcripts, overlaps between genomic loci corresponding to the probe set annotation and HP1a, Su(var)3–9 and/or H3K9me3 enriched loci listed in the relevant modENCODE database were tabulated. Probe sets annotated as transposable elements were also considered as heterochromatin sites. Non-canonical target probe sets had significantly higher proportion of such heterochromatin-related sites compared to canonical targets (p = 0.004, Fisher’s exact two-tailed test)