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. 2016 Apr 11;14(9):770–782. doi: 10.1038/cmi.2016.14

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Copyright © 2016 Chinese Society of Immunology and The University of Science and Technology

This work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by-nc-nd/4.0/

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Immunotherapies induced the desensitization of peanut-sensitized mice. Sensitized mice were EPIT-, OIT-, SLIT- or sham-treated for 8 weeks. The quantities of peanut-specific IgE (a) and IgG2a (b) were measured for each group and before treatment after sensitization (before) and after the treatment period (after). The production of Th2 cytokines (IL-5 and IL-13) (c) and IL-10 and IFN-γ (d) were measured after in vitro stimulation with peanuts for 72 h. Cytokine levels were measured using Bioplex. The data are shown as the median with range from three independent experiments with n=8 for each group of mice. *P<0.05; **P<0.01; ***P<0.001; ^#P<0.05 compared with EPIT.