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. 2017 Sep 14;12(9):e0184950. doi: 10.1371/journal.pone.0184950

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© 2017 Brown et al

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Fig 2 — (A) ddaC in wildtype larvae (GAL4-ppk1.9, UAS-mCD8GFP / +; trio^M89 / +, image shown is 300.8 μm x 650.5μm). (B) ddaC in simple trio mutant larvae (GAL4-ppk1.9, UAS-mCD8GFP / +; trio^M89 / trio^P1, image shown is 302.8 μm x 749.8μm). (C) ddaC in seq-enhanced trio mutants (GAL4-ppk1.9, UAS-mCD8GFP / seq^9.17; trio^M89 / trio^P1, image shown is 300.4 μm x 601.7μm). (D) ddaC in ppk1.9-trio rescued seq-enhanced trio mutant larvae (GAL4-ppk1.9, UAS-mCD8GFP / seq^9.17; trio^M89 / trio^P1, UAS-trio.B, image shown is 351.1 μm x 751.2μm). The dorsal midline runs horizontally through the approximate center of each image. In (A) representative examples of iso-neuronal avoidance defects are indicated with yellow triangles while representative examples of hetero-neuronal tiling defects are indicated with red triangles. Scale bars show 100 μm.