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. 2017 Sep 14;7:11619. doi: 10.1038/s41598-017-11830-9

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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sPLA₂-IIA overexpression altered the hair cycle with abnormal hair follicle morphology and hair loss. (a) Phenotypic appearance of WT, hemizygous and homozygous mice at PD21. (b) Images represents hematoxylin and eosin staining (H&E) of skin sections at various postnatal days of WT and K14-sPLA₂-IIA homozygous mice to study hair follicle cycling. (c) Graphical representation of epidermal thickness measurements are in μm. Data are presented as mean ± SD. **P < 0.005, ***P < 0.001, ****P < 0.0001. (d) Comparative analysis of hair follicle progression to different stages of hair cycle (Anagen, Catagen and Telogen) at different postnatal days in WT and K14-sPLA₂-IIA homozygous mice. (WT-Wild type, Homo- K14-sPLA₂-IIA homozygous mice, n = 3 mice/genotype. A-Anagen, C-Catagen and T-Telogen, PD-Postnatal days).