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. 2016 Mar 17;6:82–87. doi: 10.1016/j.bbrep.2016.03.008

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© 2016 The Authors

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

PMC Copyright notice

Fig. 1. — (A) The contractile reactivity to 5-HT (A) of endothelium-denuded SVs harvested from non-DM (open circles, n=9) and DM patients (closed circles, n=9). The cumulative addition of 5-HT (1 nM to 10 μM) caused SV constriction in a concentration-dependent manner in non-DM and DM groups. (B) The contractile reactivity to AngII of endothelium-denuded SVs harvested from non-DM (open circles, n=19) and DM patients (closed circles, n=8). The cumulative addition of AngII (1 nM to 10 μM) caused SV constriction in a concentration-dependent manner in the non-DM and DM groups. The data are expressed as mean±SEM. The contractile reactivity of SV to 5-HT or AngII were evaluated as the percentage of 60 mM KCl-induced vasoconstriction as the control in each SV ring. *P<0.05 compared with the non-DM group.