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. 2017 Sep 18;7:11798. doi: 10.1038/s41598-017-11276-z

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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LA and PA do not inhibit staphylokinase-mediated plasminogen activation, but inhibit plasminogen activation by recombinant streptokinase. (a) Plasminogen activation using staphylokinase. Compared to the untreated (w/o) control neither palmitoleic nor linoleic acid show an effect. (b) Plasminogen activation using recombinant cluster 2a type streptokinase. Both palmitoleic (PA) and linoleic acid (LA) (concentration for both: 40 µg/ml) inhibit plasminogen activation mediated by recombinant cluster 2a type streptokinase. w/o: untreated control. N = 3 for all experiments.