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. 2017 Sep 18;7:11811. doi: 10.1038/s41598-017-11671-6

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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The chronic administration of 24B3 did not alter the number or size of senile plaques. Representative images of immunohistochemical staining of senile plaque by 82E1 on the hippocampus of PS2Tg2576 treated with IgG (a), 82E1 (b), and 24B3 (c). The average number of senile plaques in the hippocampus (d). The average size of the senile plaques is shown in the graph (e). The numbers of mice sacrificed for staining in each group were as follows: PS2Tg2576 + IgG (n = 5), PS2Tg2576 + 82E1 (n = 6), and PS2Tg2576 + 24B3 (n = 6). *p < 0.05.