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. 2017 Jul 26;14(4):3927–3934. doi: 10.3892/ol.2017.6668

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Copyright: © Glover et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — Distribution of Bcl-2 and PCNA in various morphological lesions. (A) A significant difference in Bcl-2 was noted among the four different tissue types (P<0.0001). The pair-wise comparison showed a significant difference in Bcl-2 expression in SA vs. PIA/PAH lesions (P<0.0001); SA vs. HGPIN (P=0.012); PIA/PAH vs. HGPIN (P=0.002); and PIA/PAH vs. cancer (P=0.0009). (B) A significant difference in PCNA expression was noted among the four different tissue types (P<0.0001). The pair-wise comparison revealed a significant difference in PCNA expression in SA vs. PIA/PAH (P=0.0007); SA vs. HGPIN (P=0.002); SA vs. cancer (P=0.002); PIA/PAH vs. HGPIN (P=0.004); PIA/PAH vs. cancer (P=0.0004); and HGPIN vs. cancer (P=0.004). PCNA, proliferating cell nuclear antigen; SA, simple atrophy; PIA, proliferative inflammatory atrophy; PAH, post-atrophic hyperplasia; HGPIN, high-grade prostatic intraepithelial neoplasia; SEM, standard error of the mean.