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. 2017 Sep 20;12(9):e0184958. doi: 10.1371/journal.pone.0184958

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© 2017 Bright et al

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Fig 3 — a. NK1R binding SIDS vs. acute controls. Compared to acute controls, NK1R binding was significantly reduced in SIDS cases in the NTS (p = 0.04), DAO (p = 0.01) and MAO (p = 0.03), b. NK1R binding in SIDS vs. combined controls. NK1R binding was significantly reduced in SIDS cases in the DAO (p = 0.01) and MAO (p = 0.03) with borderline significance in the PIO (p = 0.09) when compared to all controls combined (acute, chronic and hypoxic).c. Autoradiograms displaying NK1R binding (fmol/mg) in the NTS nuclei. Absolute reductions in NK1R binding were consistently observed in SIDS cases. d. Autoradiograms displaying NK1R binding (fmol/mg) in component nuclei of the IO. NK1R binding was consistently reduced in SIDS cases. *p = <0.05, **p = <0.01.