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. 2017 Sep 20;7:12026. doi: 10.1038/s41598-017-12285-8

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Salidroside attenuates sepsis-induced lung edema and lipid peroxidation and improves PaO₂/FiO₂ ratio. Sepsis was induced through cecal ligation and puncture (CLP). (A) Lung edema was measured as the wet/dry weight ratio in mice subjected to CLP for 24 h. Salidroside (SDS 20 or 40 mg/kg) was administered to mice 30 min after CLP. (B) The PaO₂/FiO₂ ratio was detected after CLP in mice with and without salidroside treatment. (C) Malondialdehyde (MDA, lipid peroxidation marker) levels were detected in the lungs of mice subjected to CLP for 24 h. Salidroside (SDS, 20 or 40 mg/kg) was given 30 min after CLP. All data are presented as means ± SEM (n = 6 mice per group). *P < 0.05 as compared with control (vehicle). **P < 0.05 as compared with CLP alone.