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. 2017 Sep 20;7:12026. doi: 10.1038/s41598-017-12285-8

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Salidroside attenuates iNOS protein expression and NFκB-p65 phosphorylation in the lungs of mice subjected to CLP. The iNOS (A) and phospho-NFκB-p65 (B) protein expressions in the lungs were detected in mice subjected to CLP administration for 6 h. Salidroside (SDS, 20 and 40 mg/kg) was administered 30 min after CLP. Lung samples were prepared and subjected to Western blotting for protein expression and quantified by densitometry. Data are presented as mean ± SEM (n = 6). *P < 0.05 as compared with control (vehicle). **P < 0.05 as compared with CLP alone.