TABLE 1.
Diet | RA
|
O2
|
||
---|---|---|---|---|
WT | spc–mthGR | WT | spc–mthGR | |
Control | 0.87 ± 0.06 | 0.95 ± 0.04 | 0.94 ± 0.12 | 0.66 ± 0.10 |
Riboflavin | 1.28 ± 0.12# | 0.75 ± 0.15*,# | 1.43 ± 0.30*,# | 0.76 ± 0.10* |
Note. Transgenic spc–mthGR mice and WT littermates were fed either a control or high-riboflavin diet beginning at 3 weeks of age, and at 6 weeks of age were exposed to RA or >95% O2 for 84 hours. GSH concentrations were measured in 10% lung homogenates as described in Methods. Data were non-normal, and were transformed to square root and analyzed by 3-way ANOVA. An individual effect of transgene and an interaction between transgene and diet were detected. Two-way ANOVA with modified t test post hoc was performed with exposure as a fixed factor. An individual effect of transgene was detected.
Difference compared to WT, same diet.
difference between diets, same genotype; n = 5–22; P ≤ .05.