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. 2017 Sep 12;12:108–113. doi: 10.1016/j.bbrep.2017.09.001

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© 2017 The Authors

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig. 2 — Protein levels of β-tubulin, β-actin and GAPDH in the heart tissue from animal model of myocardial ischemia. (A, B) Western blot and quantitative analysis of β-tubulin, β-actin and GAPDH protein levels in heart tissue from Rhesus monkey model of early stages of myocardial ischemia (2 h after surgery, n = 2; 24 h after surgery, n = 2). (C, D) Western blot and quantitative analysis of β-tubulin, β-actin and GAPDH protein levels in heart tissue from Rhesus monkey model of myocardial infarction (4 months after surgery, n = 4). (E, F) Western blot and quantitative analysis of β-tubulin, β-actin and GAPDH protein levels in heart tissue from mouse model of different time point (1 d after surgery, n = 6; 4 d after surgery, n = 6; 7 d after surgery, n = 6). Sham: sham operated control (n = 8); IA: ischemic/ infarct area; RA: remote area. Blots were analyzed by densitometry using Fusion-Capt Software. Results are presented as means ± SD. Three independent experiments were run for each result. *: P < 0.05 versus sham operated control; #: P < 0.05.