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. 2017 Aug 24;14(9):955. doi: 10.3390/ijerph14090955

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© 2017 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Histological examination of rat livers stained with hematoxylin and eosin (H&E). (A) Control: showing normal hepatic architecture with no lesions or abnormalities; (B) CCl₄: showing congestion in the central vein associated with the infiltration of inflammatory cells; (C) T₁: showing mild hepatocytes necrosis and mononuclear cellular infiltration; (D) T₂: showing mild portal tract and lobular chronic inflammation with focal hepatocyte destruction; (E) T₃: showing that improved hepatic architecture, inflammatory cell infiltration, and hepatocytes necrosis were hardly detected (×400).

Histological examination of rat livers stained with hematoxylin and eosin (H&E). (A) Control: showing normal hepatic architecture with no lesions or abnormalities; (B) CCl₄: showing congestion in the central vein associated with the infiltration of inflammatory cells; (C) T₁: showing mild hepatocytes necrosis and mononuclear cellular infiltration; (D) T₂: showing mild portal tract and lobular chronic inflammation with focal hepatocyte destruction; (E) T₃: showing that improved hepatic architecture, inflammatory cell infiltration, and hepatocytes necrosis were hardly detected (×400).