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. 2017 Sep 26;36:133. doi: 10.1186/s13046-017-0601-6

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — RSV inhibits the proliferation of NKTCL cells. SNT-8, SNK-10 and SNT-16 cells were treated with RSV in the concentration of 0 μM (Control), 5 μM, 10 μM, 20 μM, 30 μM, 40 μM, 50 μM, 60 μM, or 70 μM for 24 h, 48 h or 72 h respectively. a The cell proliferation viability effect of RSV was measured by CCK-8 assay (n = 3). b IC₅₀ of RSV on SNT-8, SNK-10 and SNT-16 cells