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. 2016 Feb 1;3:269–278. doi: 10.1016/j.toxrep.2016.01.013

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© 2016 The Authors

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig. 2 — Cell cycle distribution (%) of SCC-61, HaCaT, SQ-20B and OSCC-3 exposed to etoposide, EGCG, GT, MT or AT for 48 h; see Section 2 for details. Values are means ± SD from three independent experiments performed in triplicate. Bars with different letters are statistically different from the control (PBS) (P < 0.05) for each cell cycle stage, □ G0/G1; S; ■ G2/M, within treatments. IC₅₀ concentrations were used and these differed depending on extract and cell line tested (see Table 1).

Inline graphic — Cell cycle distribution (%) of SCC-61, HaCaT, SQ-20B and OSCC-3 exposed to etoposide, EGCG, GT, MT or AT for 48 h; see Section 2 for details. Values are means ± SD from three independent experiments performed in triplicate. Bars with different letters are statistically different from the control (PBS) (P < 0.05) for each cell cycle stage, □ G0/G1; S; ■ G2/M, within treatments. IC₅₀ concentrations were used and these differed depending on extract and cell line tested (see Table 1).