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. 2016 Feb 7;16(2):141–149. doi: 10.1177/1533034616629261

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© The Author(s) 2016

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 3.0 License (http://www.creativecommons.org/licenses/by-nc/3.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 7. — The effects of KDM5C on the p53. A, The expression of p53, p27, and p21 in KDM5C-transfected cells was examined using Western blotting. β-Actin was used as a loading control. B, The transfection efficiency of p53 siRNA 24 hours after transfection was measured using Western blot analyses. β-Actin was used as a loading control. C, The graphs show the proliferation ability of KDM5C knockdown MKN45 and its control cells after the cells had been pretreated with p53 siRNA and its negative control. D, The summary graphs show the migration ability of KDM5C knockdown MKN45 and its control cells after the cells had been transfected with p53 siRNA and its negative control. The data represent the mean number of cells per field and are presented as the means ± SD **P < .01 versus control group; ^## P < .01 versus shKDM5C group. siRNA indicates small interfering RNA; SD, standard deviation.