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. 2017 Sep 28;8:717. doi: 10.1038/s41467-017-00769-0

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — Loss of Mef2C alters microglial immune state. a, b Representative images (a) and quantification (b) of Mef2C (red) and Iba1 (green) staining in Mef2C-KO and Mef2C-CTRL mice 2 weeks following tamoxifen-induced recombination. c–e Mean fluorescent intensity of CD45 (c), CD11b (d) and CX3CR1 (e), in Mef2C-KO and Mef2C-CTRL microglia obtained by flow cytometry analysis. In all panels **P < 0.01, ***P < 0.001; unpaired t-test, n = 8–10 per group, the results are representative of four independent experiments