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. 2017 Sep 29;7:12423. doi: 10.1038/s41598-017-12403-6

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Exosomes from irradiated cells activate the AKT-pathway to induce migration. (a) Exemplary wound healing of BHY-GFP cells after treatment with 5 µM of the AKT-inhibitor Afuresertib or DMSO, in combination with exosomes isolated from irradiated (EXO 6 Gy) and from non-irradiated cells (EXO 0 Gy). The pictures were taken 24 hours after migration start (scale bar: 500 µm). (b) Quantification of wound healing capacity with the Image Colour Analyser 24 hours [n = 3; ±SD; two-sided, paired t-test; p-value < 0.05].