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. 2017 Sep 29;8:215. doi: 10.1186/s13287-017-0673-4

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© The Author(s). 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — Alterations of molecules in the HIF-1α/apelin/APJ axis after the induction of cardiogenic differentiation. Western blot analysis (a) and qRT-PCR (b) were used to detect the expressions of molecules in the HIF-1α/apelin/APJ axis at different time points after the induction of cardiogenic differentiation. Comparisons were made between normoxia and HP groups. **p < 0.01 vs normoxia. HP hypoxia preconditioning, HIF: hypoxia-inducible factor, apelin: ligand for putative receptor protein related to angiotensin receptor AT1, APJ: putative receptor protein related to angiotensin receptor AT1