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. 2017 Sep 26;8:677. doi: 10.3389/fphar.2017.00677

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Copyright © 2017 Liu, Shang, Zhu, Yu, Qian and Duan.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Changes in the relative peak area (vs. IS) of target metabolites identified in plasma and urine (mean ± SD, n = 8). ^#p < 0.05, ^##p < 0.01 compared to the normal group. ^*p < 0.05, ^**p < 0.01 compared to the model group. PM1, lysoPE(22:4/0:0); PM2, trihydroxycholan-24-oic acid; PM3, lysoPC(17:0); PM4, lysoPE(20:2/0:0); PM5, lysoPE(24:6/0:0); PM6, taurodeoxycholic acid; PM7, arachidonic acid; PM8, tauroursocholic acid; PM9, lysoPE(22:1/0:0); PM10, chimonanthine; PM11, PG(10:0/10:0); PM12, cofaryloside; PM13, PC(19:3/0:0); PM14, lysoPE(0:0/18:0); PM15, indoxylsulfuric acid; PM16, estrone 3-glucuronide; PM17, 5-(4-acetoxybut-1-ynyl)-2,2′-bithiophene; PM18, polyethylene oxidized; PM19, succinoadenosine; PM20, 17-hydroxypregnenolone sulfate; PM21, uralenneoside; PM22, 2,8-dihydroxyquinoline-beta-D-glucuronide; PM23, dihydrocaffeic acid 3-sulfate.