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. Author manuscript; available in PMC: 2017 Oct 1.
Published in final edited form as: J Dev Orig Health Dis. 2016 Dec 28;8(2):155–160. doi: 10.1017/S2040174416000775

Fig. 1.

Fig. 1

Effects of prenatal nicotine exposure on synaptic proteins of CA3. (a) NMDAR subunits GluN1, GluN2A, GluN2B, GluN2C, (b) AMPAR subunits GluA1 and GluA2 and (c) soluble postsynaptic proteins, PSD95, CaMKIIIa, CaM and presynaptic SNAP25, quantified after Western blotting. *,P < 0.05; n = 4–10. NMDAR, N-methyl-D-aspartate; AMPAR, α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor.