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. Author manuscript; available in PMC: 2017 Oct 1.
Published in final edited form as: Psychosom Med. 2015 Jul-Aug;77(6):616–621. doi: 10.1097/PSY.0000000000000207

FIGURE 2.

FIGURE 2

Comparison of neurochemical profiles of patients with T1DM (n = 13) relative to nondiabetic controls (n = 32). Metabolite concentrations were measured from the gray matter–rich occipital lobe (A) and white matter–rich parieto-occipital region (B). Error bars indicate significance levels: *p < .05 and **p < .01. MM is quantified in arbitrary units. T1DM = Type 1 diabetes; MM = macromolecules; Asp = aspartate; Asc = ascorbate; Cr = creatine; GABA = g-aminobutyric acid; Glc = glucose; Gln = glutamine; Glu = glutamate; GSH = glutathione; myo-Ins = myo-inositol; scyllo-Ins = scyllo-inositol; Lac = lactate; NAA = N-acetylaspartate; NAAG = N-acetylaspartylglutamate; PE = phosphoethanolamine; Tau = taurine; GPC+PC = the sum of glycerophosphocholine and phosphocholine; Cr = creatine; PCr = phosphocreatine. Reprinted by permission from Macmillan Publishers Ltd: Journal of Cerebral Blood Flow and Metabolism (18).

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