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. 2017 Sep 27;10:308. doi: 10.3389/fnmol.2017.00308

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Copyright © 2017 Zhu, Chen, Meng, Ju, Zhang, Wu, Guo and Tian.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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CRH is a target of miR-142 and miR-376c. CRH protein expression (A) in primary hypothalamus neurons transfected with miR-142, miR-200c, miR-325, miR-361, miR-376c and miR-429 agomir or the negative control. The data are presented as the mean ± SD (n = 5) *vs. miR-NC-OE group (p < 0.05). The binding site (B) of miR-142, miR-200c and miR-376c on the 3′-untranslatedregions (3′ UTR) of CRH and their mutant plasmid. The luciferase activity (C) of the miR-142, miR-200c, miR-376c onto the 3′ UTR of CRH. The data are presented as the mean ± SD (n = 4) *vs. miR-NC-OE group (p < 0.05).