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. 2017 Oct 4;7:12653. doi: 10.1038/s41598-017-12929-9

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Glucose uptake in 58As9 cells with or without YC-1 + GI. (a,b) 2DG uptake levels in control and YC-1-treated cells were analyzed under normoxia (a) and hypoxia (b) for 24 h in the presence or absence of insulin. (c) Western blot analysis of membranous GLUT1 (54 kDa) levels in control and YC-1-treated cells that were cultured under hypoxia for 24 h in the presence or absence of G and/or I. β-actin was equally expressed in all cells. ns: not significant, **p < 0.01.