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. 2017 Oct 4;17:86. doi: 10.1186/s12935-017-0457-z

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© The Author(s) 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — Vemurafenib induces a feedback activation of EGFR phosphorylation in BRAF V600E thyroid carcinoma cells. a–c EGFR, phosphoEGFR, AKT, phosphoAKT, ERK and phosphoERK immunoblot analysis in BRAF V600E FRO (a), BHT101 (b) and BCPAP (c) thyroid carcinoma cell lines exposed to 10 μM PLX4032 for 4–8, 15 and 24 h