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. 2017 Oct 5;7:12711. doi: 10.1038/s41598-017-12637-4

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Patient pathways (for 40 patients between March – October 2015) and whole genome sequence Maximum Likelihood tree. The WGS tree supports the emergence and clonal spread of colistin resistance caused by two different mechanisms (MgrB: C16 and MgrB:K3), and also the emergence of a third type of colistin resistance (PhoQ:L396Q) that did not spread clonally. Red colour in tree = colistin resistant isolates; Black colour in tree = colistin susceptible isolates. Isolates KP_NDM_1 and KP_NDM_2 were first identified in July and December 2014, respectively.