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. 2017 Oct 6;8:804. doi: 10.1038/s41467-017-00935-4

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Overexpression of Pot1a maintains HSC LTR activity. a, b Results of primary BMT. a Percentage of donor-derived (Ly5.1⁺GFP⁺) cells in recipient mice PB, BM LSK and LT-HSCs 4 months after BMT. b Peripheral blood analysis of B, T, and myeloid cell lineages 4 months post BMT. Data are expressed as the mean ± SD (n = 5 group^–1, **p < 0.05 by t-test). Representative data from two independent experiments are shown. c, d Results of secondary BMT. c The percentage of donor-derived (Ly5.1⁺GFP⁺) cells in recipient mice PB, BM LSK and LT-HSCs 4 months post secondary BMT. d Peripheral blood analysis of B, T, and myeloid cell lineages 4 months post secondary BMT. Data are expressed as the mean ± SD (n = 5, *p < 0.01 by t-test). Representative data from two independent experiments are shown