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. 2017 Aug 2;31(11):5122–5132. doi: 10.1096/fj.201700317R

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This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 International (CC BY-NC 4.0) (http://creativecommons.org/licenses/by-nc/4.0/) which permits noncommercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 1. — GR^LysMCre mice demonstrated increased mortality rates as a result of LV rupture and exacerbated cardiac remodeling and function after myocardial infarction. A) Kaplan-Meier survival curve. B) Representative LV pressure–volume loops measured in vivo with conductance catheter in GR^flox and GR^LysMCre mice 7 d after sham operation (sham) or MI. C) Representative sections from GR^flox and GR^LysMCre infarcted hearts and infarct size. D) LV end-systolic and end-diastolic volumes, LV maximal rate of pressure rise (LV dP/dt_max), maximal rate of pressure decline (LV dP/dt_min), and LV dP/dt_max divided by instantaneous pressure (IP) and LV ejection fraction. Means ± sem (n = 10; male/female: 3/7). *P < 0.01 vs. GR^flox.