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. 2017 Oct 11;7:13004. doi: 10.1038/s41598-017-12532-y

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Functional similarity between mouse MRCS1 and Xenopus MFCS4. LacZ expression in the teeth buds of the maxilla (a–c) and papillae of the tongue (d–i). Expression directed by the whole fragments of mouse MRCS1 (a,d), mouse MFCS4 (b,e), Xenopus MFCS4 (c,f) or three tandem copies of the core sequence of mouse MRCS1 (g), mouse MFCS4 (h) or Xenopus MFCS4 (i). (j) Schematic diagram of Shh regulation by MRCS1 (MR) and MFCS4 (M4). MRCS1 primarily regulates Shh in the oral epithelium of mouse. MFCS4 alone regulates Shh in Xenopus.