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. 2017 Oct 12;8:890. doi: 10.1038/s41467-017-01026-0

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Driver landscape of 104 chordomas. a For the discovery cohort, in every tumour (column), driver events are indicated by gene (row) and type of mutation (colour coded; refer to legend). For extension cases, the overall prevalence of driver events in each gene is indicated (colour coded by mutation class). b The LYST protein is depicted, indicating the position of truncating mutations in the discovery (red circles) and extension (red squares) samples. Functional domains are indicated in blue