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. 2017 Aug 16;14(4):3720–3726. doi: 10.3892/etm.2017.4940

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Copyright: © Xu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 6. — Knockdown of TCTN1 induced alteration in cell cycle and apoptotic regulator expression in thyroid cancer cells. Expression levels of Bad, Bcl-2, caspase-3 and PARP were analyzed with Western blotting in CAL62 (A) and 8305C (B) cells after infected with shCon or shTCTN1. Expression levels of Cyclin B1, CDK2, Cyclin A2 and Cyclin D1 were determined in CAL62 (C) and 8305C (D) cells after infected with shCon or shTCTN1 using western blotting. GAPDH was used as an internal control.