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. 2017 Oct 13;9:82. doi: 10.1186/s13098-017-0282-5

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© The Author(s) 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — Representative photomicrographs of liver sections stained with Hematoxylin & Eosin (a–c), Sudan III (d–f) and Naphtol AS-D (g–i) in the liver in the control group (C); diabetic and high fat diet mice (HDC); diabetic and high fat diet mice treated with aliskiren (HDA) (× 400 magnification). Graphic representation of steatosis score (j), Sudan III labeling (k), inflammation score (l) and neutrophil influx (m) in C, HDC and HDA groups. *P < 0.05, C × HDC; ^●P < 0.05, HDC × HDA. (n = 4–6 mice per group)