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Expression of BCR kinases are maintained by the chaperon function of HSP90. (a) Cells were treated with 0.5 μM PU-H71 for the indicated hours. Protein lysates were probed for BCR signaling molecules. GAPDH, loading control. (b) Cells were treated with PU-H71 at indicated concentrations for 48 h. (c) Cells co-cultured with HS-5 or NKTert were treated with PU-H71 at indicated concentrations for 48 h.
