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. 2017 Jul 24;16(4):3901–3909. doi: 10.3892/mmr.2017.7078

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Copyright: © Jiang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — Histological examination of carbon tetrachloride-induced liver fibrosis in rats at 12 weeks. Magnification, ×200. (A) HE and (B) Masson staining of liver tissue from the control group. (C) HE and (D) Masson staining of liver tissue from the model group. (E) HE and (F) Masson staining of liver tissue from the SGJPF group. Arrows: a, increased fatty degeneration; b, inflammatory cell infiltration; c, a dissolved nucleus; d, increased hyperplastic collagen fibers. HE, hematoxylin and eosin; SGJPF, Shu Gan Jian Pi formula.