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. 2017 Aug 10;16(4):5441–5449. doi: 10.3892/mmr.2017.7219

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Copyright: © Wang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 8. — Treatment with angelicin reduced the proliferation and induced the apoptosis of tumor cells, and inhibited angiogenesis in vivo. (A) Representative photomicrographs of immunohistochemical staining for TUNEL, Ki-67 and p-VEGFR2 in tumor tissue samples; magnification, ×200. Quantitative results of immunohistochemical staining for (B) TUNEL, (C) Ki-67 and (D) p-VEGFR2. Data are expressed as the mean ± standard deviation of 6 individual mice in each group from one experiment. *P<0.05, **P<0.01 vs. the NC group. TUNEL, terminal deoxynucleotidyl transferase dUTP nick-end labeling; p-VEGFR2, phosphorylated vascular endothelial growth factor receptor 2; NC, negative control; IOD, integrated optical density.