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. Author manuscript; available in PMC: 2018 Oct 1.
Published in final edited form as: Biochim Biophys Acta. 2017 Jun 15;1863(10 Pt B):2654–2660. doi: 10.1016/j.bbadis.2017.06.008

Table 1.

HR = heart rate; IVSd = diastolic interventricular septum thickness; LVIDs = systolic left ventricular internal dimension; LVIDd = diastolic left ventricular internal dimension; FS = left ventricular fractional shortening; LVM = left ventricular mass; LVPWd = diastolic left ventricular posterior wall thickness.

Before CLP After CLP
Non-obese Obese Non-obese Obese
HR (bpm) 442 ± 85 437 ± 24 325 ± 46* 430 ± 25#
IVSd (mm) 0.85 ± 0.1 0.78 ± 0.02 1 ±0.16 0.86 ± 0.15
LVIDs (mm) 2.53 ± 0.35 3.17 ± 0.41# 2.27 ± 0.85 2.54 ± 0.34*
LVIDd (mm) 4 ± 0.41 4.20 ± 0.35 3.51 ± 0.76 3.68 ± 0.4*
FS (%) 34 ±4 25 ± 5# 37 ± 10 31 ± 4*
LVM (mg) 96 ± 14.1 95.2 ± 14.8 91 ± 8 84 ± 7*
LVPWd (mm) 0.74 ± 0.07 0.73 ± 0.1 0.88 ± 0.23 0.77 ± 0.07

HR = heart rate; IVSd = diastolic interventricular septum thickness; LVIDs = systolic left ventricular internal dimension; LVIDd = diastolic left ventricular internal dimension; F5 = left ventricular fractional shortening; LVM = left ventricular mass; LVPWd = diastolic left ventricular posterior wall thickness;.

#

p<0.05 versus non-obese by t-test,

*

p<0.05 versus before CLP by paired t-test. n=8 mice/group.