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. 2017 Oct 23;7:13850. doi: 10.1038/s41598-017-14120-6

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Early-onset and classical forms of type 2 diabetes generate a gene signature. (a) Venn diagram depicting genes differentially expressed in muscle from type 2 diabetic patients compared with muscle from age- and BMI-matched nondiabetic subjects. (b) Heatmap of genes differentially expressed in muscle from type 2 diabetic patients. YT2 are early-onset type 2 diabetic subjects; OT2 are older type 2 diabetic subjects, and their respective controls are YC and OC. (c) GSEA of genes dysregulated in muscles from type 2 diabetic patients.