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. 2017 Oct 19;8:1345. doi: 10.3389/fimmu.2017.01345

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Copyright © 2017 Jia, Shan, Pan, Feng, Lv, Sun, Li, Wu, Zhang, Chen, Diana, Sun and Chen.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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CB0313.1 restores the diabetes-induced gut microbial dysbiosis. (A,B) Abundance of the most important phyla in each group. Abundance of the main altered classes (C), orders (D), families (E), and genera (F) in each group. (G) Principal coordinate analysis (PCoA) plot of weighted UniFrac distances, each dot representing a feces community; the percentage of variation explained by each principal coordinate is shown in parentheses. (H) Relative abundance of CB0313.1. Predominant butyrate producing genes: relative abundance of butyrate kinase (buk) and butyryl-CoA DNA in feces (I,J). (K) SCFA concentration in feces measured by GC-MS. Analysis of variance followed by the indicated post hoc test was performed to determine the significance among the three groups. Data are mean ± SEM (n = 8 mice per group). *, **, *** p < 0.05, p < 0.01, p < 0.001 vs NOD control mice by t-test.