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. 2017 Oct 20;7:13663. doi: 10.1038/s41598-017-13961-5

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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How the cross-talk mitochondrion-nucleus may be affected. Flow cytometric analysis of some key factors of the intracellular network confirmed the increased phosphorylation of CREB and revealed a parallel higher phosphorylation of FAK which, together with the increased expression of PGC1a p53 and PPARg, testified for the increased mitogenesis in an attempt to improve mitocohondrial activitiy. Both MFI and percentage of positive cells were increased in VHM population. Results are referred to analyses carried out in basal condition. Data are mean ± SD of all the performed experiments. *p = 0.05; **p = 0.01.