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. 2017 Jun 30;26(7):1276–1285. doi: 10.1177/0963689717716510

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© The Author(s) 2017

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (http://www.creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 3. — The expression of Trim32 at 7 d after traumatic brain injury (TBI) in mice. (a) Reverse transcription polymerase chain reaction (RT-PCR) results in the injury cortex and sham-operated mice. β-Actin was used as control. (b) This is to exhibit the protein level of Trim32. β-Actin was used as control. Lane 1 in the left side displays the protein band in the sham group, and lane 2 shows the protein band at 7 d postoperation. The relative translation of Trim32 was higher at 7 d postoperation. *P < 0.05 compared with the sham group.