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. 2017 Jun 30;26(7):1276–1285. doi: 10.1177/0963689717716510

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© The Author(s) 2017

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (http://www.creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 7. — The expression of p73 at 7 d after traumatic brain injury (TBI) from TBI mice and Trim32 knockout mice (Trim32-KO [+/−]) TBI mice. (A) Reverse transcription polymerase chain reaction (RT-PCR) results of p73 in TBI mice and Trim32 KO (+/−) TBI mice. β-Actin was used as control. (B) This is to exhibit the protein level of p73. β-Actin was used as control. Lane 1 in the left side displays the protein band in the sham group, lane 2 shows the protein band in wild-type (WT)-TBI mice, and lane 3 shows the protein band in Trim32 KO (+/−) TBI mice. The relative translation of p73 was lower in Trim32 KO (+/−) TBI mice. *P < 0.05 compared with the sham group; #P < 0.05 compared with the WT-TBI group.