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. 2017 Oct 26;7:14143. doi: 10.1038/s41598-017-14396-8

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Effect of neonatal hyperoxia on PAC number and function. Number of PACs isolated from the bone marrow (A) and % of PACs in the spleen as assessed by flow cytometry (B) in mice exposed to neonatal hyperoxia or normoxia. To assess PAC adhesion (C), PACs were labelled with a DiI fluorescent marker and allowed to adhere to a monolayer of tumor necrosis factor-α-stimulated HUVECs. Data are mean ± SEM (n = 4–6/group). *P < 0.05 vs. normoxia.