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. 2017 Jun 23;2:17022. doi: 10.1038/sigtrans.2017.22

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Copyright © 2017 The Author(s)

This work is licensed under a Creative Commons Attribution 4.0 International License. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/

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AT2R expression in white adipocyte is upregulated by thyroid hormone T3. Human white adipocytes (day 14) were treated without (−) (control) or with (+) AngII, ZD7155, PD123319, 1 μM CL316,243 or 50 nM thyroid hormone T3 for 4–5 days. (a–d) The representative immunoblots of protein expressions (UCP1, CITED1, PRDM16, AT1R (~41 kDa), AT2R (~43 kDa), β1-adrenergic receptor (β1AR, ~65 kDa), β3AR (~44 kDa), thyroid receptor α1 (TRα1, ~48 kDa), TRβ1 (~53 kDa), aP2 and actin) and the statistics (mean±s.e.m., n=4; normalized to actin densities) are shown accordingly. (e) The representative confocal images of immunostained AT1R, AT2R, TRα and TRβ1 in differently treated white adipocytes (from three separate experiments). Scale bars=10 μm. *P<0.05, **P<0.01 versus control;^#P<0.05 between indicated pairs.