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. 2017 Nov 1;7:14876. doi: 10.1038/s41598-017-13974-0

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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TUDCA treatment improves glucose tolerance, insulin sensitivity and insulinemia in HFD mice. Blood glucose during ipGTT (A) and ipITT (C). Area under the curve (AUC) of total blood glucose concentration during ipGTT (B) and glucose disappearance rate during ipITT (KITT) (D). Plasma insulin in fed (E) and fasting (F) state. Mice were fed a control diet (CON and CON + TUDCA) or high fat diet (HFD and HFD + TUDCA) for 12 weeks, and received or not i.p. 300 mg/kg TUDCA during 15 days, as indicated. Data are mean ± SEM (n = 4–8). *P ≤ 0.05 vs CON.