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. 2017 Nov 1;7:14793. doi: 10.1038/s41598-017-14933-5

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Sea bass (A) hepcidin expression in the liver and (B) ferroportin expression in the liver and intestine under experimental anemia. Fish were made anemic by removing 2% v/w body mass of blood through the caudal vessels (n = 4). Values are expressed as mean fold change ± SD. Actb was used as the housekeeping gene. Differences from the control groups were considered significant at *p < 0.05, **p < 0.01, and ***p < 0.001.